Saint-Denis, France

Emmanuel Philippe Dassa

USPTO Granted Patents = 1 


Average Co-Inventor Count = 5.0

ph-index = 1


Company Filing History:


Years Active: 2009

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1 patent (USPTO):Explore Patents

Title: Emmanuel Philippe Dassa: Innovator in Mitochondrial Research

Introduction

Emmanuel Philippe Dassa is a notable inventor based in Saint-Denis, France. He has made significant contributions to the field of mitochondrial research, particularly through his innovative patent related to alternative oxidase.

Latest Patents

Dassa holds a patent titled "Alternative oxidase and uses thereof." This invention pertains to a method for combating disorders that affect the mitochondrial oxidative phosphorylation system. The method involves the allotopic expression of the cyanide-insensitive alternative oxidase (AOX) in human cells. The successful expression of AOX in human cells has demonstrated remarkable cyanide resistance to mitochondrial substrate oxidation. It also alleviates oxidative stress, reduces apoptosis susceptibility, and mitigates metabolic acidosis. AOX is well tolerated when expressed throughout the entire organism, making it a valuable tool to limit the harmful consequences of respiratory chain deficiency.

Career Highlights

Throughout his career, Dassa has worked with various organizations, including Licentia Ltd. and the Institut National de la Santé et de la Recherche Médicale. His work has focused on advancing the understanding of mitochondrial function and developing therapeutic strategies to address related disorders.

Collaborations

Dassa has collaborated with esteemed colleagues in his field, including Pierre Jean Frederic Rustin and Howard T Jacobs. These collaborations have further enriched his research and contributed to the advancement of mitochondrial science.

Conclusion

Emmanuel Philippe Dassa's innovative work in mitochondrial research, particularly through his patent on alternative oxidase, showcases his commitment to advancing medical science. His contributions have the potential to significantly impact the treatment of mitochondrial disorders.

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