Sherbrooke, Canada

Raymund Wellinger


 

Average Co-Inventor Count = 2.0

ph-index = 1

Forward Citations = 6(Granted Patents)


Company Filing History:


Years Active: 2002

Loading Chart...
Loading Chart...
1 patent (USPTO):Explore Patents

Title: Raymund Wellinger: Innovator in Telomere Research

Introduction

Raymund Wellinger is a notable inventor based in Sherbrooke, Canada. He has made significant contributions to the field of molecular biology, particularly in understanding telomere dynamics. His work focuses on the mechanisms that influence telomere length and their implications for cellular aging and proliferation.

Latest Patents

Wellinger holds a patent titled "Methods for monitoring the binding of A1/UP1 to single-stranded nucleic acid sequences, and to measure the effect of this binding on telomere extension and protection." This invention addresses the critical role of telomeres in cellular function. It explores how hnRNP A1 and its derivative UP1 can affect telomere length by binding to telomerase RNA and protecting mammalian telomeric DNA. The patent also discusses agents that can interfere with this binding, providing a framework for assessing their biological impact.

Career Highlights

Throughout his career, Wellinger has worked with various organizations that focus on biotechnology and pharmaceuticals. Notable companies include Telogene Inc. and Gemin X Pharmaceuticals Canada Inc. His research has contributed to advancements in understanding telomere biology and its relevance to health and disease.

Collaborations

Wellinger has collaborated with several professionals in his field, including Benoit Chabot. These partnerships have enhanced the scope and impact of his research.

Conclusion

Raymund Wellinger is a prominent figure in telomere research, with a patent that highlights his innovative approach to understanding cellular aging. His contributions continue to influence the field of molecular biology and have potential implications for therapeutic developments.

This text is generated by artificial intelligence and may not be accurate.
Please report any incorrect information to support@idiyas.com
Loading…