Company Filing History:
Years Active: 2014-2017
Title: Akiomi Tanaka: Innovator in Neuropathic Pain Treatment
Introduction
Akiomi Tanaka is a notable inventor based in Tokyo, Japan. He has made significant contributions to the field of neuropathic pain treatment, holding two patents that focus on innovative solutions for improving nerve-related dysfunctions. His work aims to provide alternatives to existing treatments, minimizing adverse reactions and enhancing patient outcomes.
Latest Patents
Tanaka's latest patents include an "Agent for Neuropathic Pain" and a "Method for Treating Neuropathic Pain." The first patent addresses the need for a substance that acts as an active ingredient for improving neuropathic pain through a unique mechanism of action. This agent is characterized by its use of a lyase enzyme, specifically chondroitinase ABC, which selectively degrades chondroitin sulfate chains in proteoglycans. The second patent focuses on an improving agent for dysfunction caused by nerve damage, utilizing an endo-β-N-acetylglucosaminidase type enzyme to hydrolyze N-acetylglucosamide bonds in keratan sulfate. This innovative approach has shown promise in achieving clinical improvement in both motor and sensory neuron dysfunctions.
Career Highlights
Throughout his career, Akiomi Tanaka has worked with esteemed institutions such as Nagoya University and Seikagaku Corporation. His experience in these organizations has allowed him to develop and refine his inventions, contributing to advancements in medical treatments for neuropathic pain.
Collaborations
Tanaka has collaborated with notable colleagues, including Yukihiro Matsuyama and Kenji Kadomatsu. These partnerships have fostered a collaborative environment that encourages innovation and the sharing of ideas in the field of neuropathic pain research.
Conclusion
Akiomi Tanaka's work in developing innovative treatments for neuropathic pain showcases his dedication to improving patient care. His patents reflect a commitment to finding effective solutions that address the complexities of nerve damage and dysfunction.