The patent badge is an abbreviated version of the USPTO patent document. The patent badge does contain a link to the full patent document.

The patent badge is an abbreviated version of the USPTO patent document. The patent badge covers the following: Patent number, Date patent was issued, Date patent was filed, Title of the patent, Applicant, Inventor, Assignee, Attorney firm, Primary examiner, Assistant examiner, CPCs, and Abstract. The patent badge does contain a link to the full patent document (in Adobe Acrobat format, aka pdf). To download or print any patent click here.

Date of Patent:
Sep. 12, 2017

Filed:

Aug. 14, 2015
Applicants:

Cornell University, Ithaca, NY (US);

The Rockefeller University, New York, NY (US);

Inventors:

Timothy Vartanian, New York, NY (US);

Kareem Rashid Rumah, New York, NY (US);

Vincent A. Fischetti, Hempstead, NY (US);

Assignees:

Cornell University, Ithaca, NY (US);

The Rockefeller University, New York, NY (US);

Attorney:
Primary Examiner:
Int. Cl.
CPC ...
A61K 31/7048 (2006.01); A61K 38/47 (2006.01); A61K 39/00 (2006.01); C07K 16/12 (2006.01); A61K 35/741 (2015.01); A61K 35/742 (2015.01); C12Q 1/68 (2006.01); G01N 33/569 (2006.01);
U.S. Cl.
CPC ...
C07K 16/1282 (2013.01); A61K 31/7048 (2013.01); A61K 35/741 (2013.01); A61K 35/742 (2013.01); A61K 38/47 (2013.01); A61K 39/00 (2013.01); C12Q 1/689 (2013.01); G01N 33/56911 (2013.01); A61K 2039/505 (2013.01); C07K 2317/76 (2013.01); C12Q 2600/118 (2013.01); C12Q 2600/142 (2013.01); C12Y 302/01017 (2013.01); G01N 2333/33 (2013.01); G01N 2469/20 (2013.01); G01N 2500/04 (2013.01); G01N 2500/10 (2013.01); G01N 2800/285 (2013.01);
Abstract

The invention provides epsilon toxin (ETX) produced bytype B or type D as a causative toxin for human multiple sclerosis (MS). The invention further identifies ETX binding receptor MAL for ETX mediated cell death and other toxin-logical activities in MS. Methods and compositions to prevent humans from multiple sclerosis (MS) and/or treating MS by directly or indirectly interfering with epsilon toxin (ETX), its binding receptor (e.g., MAL), or ETX-receptor interactions so as to inhibit or suppress downstream ETX mediated receptor signaling activities are provided. Also provided are various methods to detect, diagnose, monitor, assess multiple sclerosis (MS) by determining an expression level of ETX gene or its encoding protein in human patient suspected for and/or at risk for multiple sclerosis (MS).


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