The patent badge is an abbreviated version of the USPTO patent document. The patent badge does contain a link to the full patent document.
The patent badge is an abbreviated version of the USPTO patent document. The patent badge covers the following: Patent number, Date patent was issued, Date patent was filed, Title of the patent, Applicant, Inventor, Assignee, Attorney firm, Primary examiner, Assistant examiner, CPCs, and Abstract. The patent badge does contain a link to the full patent document (in Adobe Acrobat format, aka pdf). To download or print any patent click here.
Patent No.:
Date of Patent:
Jun. 15, 2010
Filed:
Aug. 25, 2008
Michael E. Brune, Mundelein, IL (US);
Peer B. Jacobson, Libertyville, IL (US);
Jeffrey J. Rohde, Evanston, IL (US);
Jiahong Wang, Lake Bluff, IL (US);
Steven Fung, Mount Prospect, IL (US);
Ravi Kurukulasuriya, East Lyme, CT (US);
David J. Madar, Gilbert, AZ (US);
James T. Link, Stanford, CA (US);
Vince S. Yeh, San Diego, CA (US);
Michael E. Brune, Mundelein, IL (US);
Peer B. Jacobson, Libertyville, IL (US);
Jeffrey J. Rohde, Evanston, IL (US);
Jiahong Wang, Lake Bluff, IL (US);
Steven Fung, Mount Prospect, IL (US);
Ravi Kurukulasuriya, East Lyme, CT (US);
David J. Madar, Gilbert, AZ (US);
James T. Link, Stanford, CA (US);
Vince S. Yeh, San Diego, CA (US);
Abbott Laboratories, Abbott Park, IL (US);
Abstract
Methods for treating a mammal suffering from glucocorticoid-related diseases and conditions, comprising administering to the mammal an effective amount of a selective inhibitor of 11-beta-hydroxysteroid dehydrogenase Type 1 enzyme activity, wherein the inhibitor is a compound of formula (I): or a pharmaceutically acceptable salt, prodrug, salt of a prodrug, or a combination thereof. Methods of inhibiting 11-beta-hydroxysteroid dehydrogenase Type 1 enzyme. Methods of treating neuronal degeneration, dysfunction, acute psychosis, anxiety, dementia, depression, non-insulin dependent type 2 diabetes, insulin resistance, obesity, lipid disorders, metabolic syndrome and other diseases and conditions that are mediated by excessive glucocorticoid action.