The patent badge is an abbreviated version of the USPTO patent document. The patent badge does contain a link to the full patent document.

The patent badge is an abbreviated version of the USPTO patent document. The patent badge covers the following: Patent number, Date patent was issued, Date patent was filed, Title of the patent, Applicant, Inventor, Assignee, Attorney firm, Primary examiner, Assistant examiner, CPCs, and Abstract. The patent badge does contain a link to the full patent document (in Adobe Acrobat format, aka pdf). To download or print any patent click here.

Date of Patent:
Mar. 02, 2010

Filed:

Jun. 19, 2007
Applicants:

Ryukou Tokuyama, Tokyo, JP;

Toshifumi Wakamatsu, Tokyo, JP;

Tatsurou Ichige, Tokyo, JP;

Susumu Muto, Tokyo, JP;

Akiko Itai, Tokyo, JP;

Inventors:

Ryukou Tokuyama, Tokyo, JP;

Toshifumi Wakamatsu, Tokyo, JP;

Tatsurou Ichige, Tokyo, JP;

Susumu Muto, Tokyo, JP;

Akiko Itai, Tokyo, JP;

Attorney:
Primary Examiner:
Assistant Examiner:
Int. Cl.
CPC ...
A01N 43/42 (2006.01); A61K 31/44 (2006.01); C07D 241/04 (2006.01); C07D 295/00 (2006.01);
U.S. Cl.
CPC ...
Abstract

A compound represented by the following formula (I) or a salt thereof: wherein R, R, Rand Rrepresent hydrogen atom, a halogen atom, cyano group, nitro group, a Calkyl group, a halogenated Calkyl group or a Calkoxy group, Rrepresents a halogen atom, cyano group, a Calkyl group, a halogenated Calkyl group or a Calkoxy group, Rrepresents a Ccycloalkyl group, a substituted Ccycloalkyl group, a 5 to 7-membered completely saturated heterocyclic group or a substituted 5 to 7-membered completely saturated heterocyclic group, X represents a single bond, oxygen atom, sulfur atom, NR, —O—CH— or —N(R)—CH—, Rrepresents hydrogen atom or a Calkyl group, or Rmay combine with a substituent of Rto represent a single bond, methylene group or ethylene group, Rrepresents hydrogen atom, a Calkyl group or a Caralkyl group, which is useful as an active ingredient of a medicament for prophylactic and/or therapeutic treatment of diseases caused by an activation of STAT6 and/or NF-κB.


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