Hyundai Motor Company

Snu Rdb Foundation

Industry-Academic Cooperation Foundation, Chosun University

Kia Motors Corporation

Korea Research Institute of Bioscience and Biotechnology

Industry-academic Cooperation Foundation, the Catholic University of Korea

Samsung Electronics Co., Ltd.

Kia Motor Corporation

Ho Youn Kim

Young Sik Kim

Ho Jin Jung

Jong Seon No

Hyun Soo Ahn

Jun Young Woo

Kang Seok Lee

Young Woo Park

Jung Jae Yu

Mi La Cho

Ki Won Jo

Byoung Wook Lee

Ho Ryong Yoo

Jun Geol Ryu

Jin Sil Park

Jae Kyeong Byun

Yun Ju Woo

Srok Ho Yoo

Jong-Ho Lee

Eun Kyung Lee

Mi-La Cho

Chung Hi Lee

Mi Kyung Park

Young Sik Moon

Hye Jwa Oh

Eun Jung Song

Sun-Ha Yoon

Ji Hyun Park

Dong Jin Kim

Min Ji Seo

Joon-Goo Jung

Myung-ho Sohn

Kyu Won Cho

Jae Won Jeon

Young-Soon Jang

Sun Jung Cho

Jin Mi Oh

Ji-Hun Moon

Jong Yoon Yoon

Hye In Choi

A memory device includes; a memory module including a memory array, and a memory controller that retrieves read data from memory cells of the memory array. The memory controller includes a fault detector that detects faulty addresses associated with faulty memory cells among the memory cells providing data errors. The fault detector includes; a first inverter that generates inverted read data by reading and inverting the read data, wherein the inverted read data is stored in the memory array, a first buffer that stores the read data and provides buffered data, an XOR operator that receives the buffered data from the first buffer, receives read-out inverted data generated by reading the inverted read data stored in the memory array, and performs an XOR operation on the buffered data and the read-out inverted read data to generate calculation data, a fault address detection unit that identifies the faulty addresses in response to the calculation data and generates faulty address information, a second inverter that generates inverted read-out inverted read data by receiving and inverting the read-out inverted read data, and an error pattern change unit that converts an uncorrectable error (UE)-causing data into a correctable error (CE)-causing data.

Memory controller and memory device including the same

A method of performing cross-authentication in a vehicle controller interworking with an external device includes: generating a random number S and transmitting the random number S to the external device according to an authentication request message received from the external device; generating a variable i using a first function having the random number S as a parameter; generating a first session key Ks using a second function having the variable i and a pre-stored secret key K as parameters; receiving a first response key from the external device; generating a second response key using a third function having the random number S, the variable i and the first session key Ks as parameters; and authenticating the external device based on whether the first response key is equal to the second response key.

Method and apparatus for performing cross-authentication based on secret information

A method and apparatus for authenticating packets in a controller area network (CAN) are disclosed. The method includes transmitting messages using a mixture of message authentication codes (MACs) in a controller area network (CAN). In addition, a first MAC is generated using a first message and the first MAC is divided into a first MAC part and a second MAC part. A second MAC is generated using a second message and the second MAC is divided into a third MAC part and a fourth MAC part. A linear operation is performed between the second MAC part and the third MAC part to generate a first authentication MAC. The first message is transmitted with the first MAC part and the second message is transmitted with the first authentication MAC.

Method of authenticating can packets using mixture of MACs and apparatus for implementing the same

A method of transmitting K messages using divided message authentication codes (MACs) in a controller area network (CAN) includes: generating a MAC using a first message and a specific MAC, performing a first operation with respect to j using j-th messages subsequent to the first message and a second MAC part of the generated MAC, performing a second operation with respect to j using a result of the performed first operation and a j-th subblock subsequent to a first MAC subblock among K MAC subblocks obtained by dividing a first MAC part of the generated MAC, transmitting the first message along with the first MAC subblock, and transmitting K-1 j-th messages in an order of j, each of the j-th messages being transmitted along with a j-th result of the performed second operation.

Method of sequentially authenticating CAN packets using divided MACS and apparatus for implementing the same

The present invention relates to TNFR2-IL21R fusion protein acting as a double-antagonist to TNF-alpha (&#x3b1;) and IL-21. The composition containing the double antagonist to TNF-&#x3b1; and Il-21 (TNFR2-IL21R fusion protein), known as major causes of autoimmune rheumatoid arthritis, one of autoimmune diseases, can reduce the secretion of inflammatory cytokine, increase the secretion of anti-inflammatory cytokine, and suppress the differentiation of osteoclasts better than single proteins such as TNFR2-Fc and IL21R-Fc. The TNFR2-IL21R fusion protein of the present invention has not only excellent treatment effect on arthritis in CIA mouse model not also excellent treatment effect on autoimmune rheumatoid arthritis by increasing the expression of Treg, the immune suppressive cells. Therefore, the TNFR2-IL21R fusion protein of the present invention can be effectively used as an active ingredient for the composition for the prevention and treatment of autoimmune disease.

Dual antagonist for TNF-&#x3b1; and IL-21 for preventing and treating autoimmune diseases

The present invention relates to TNFR2-TWEAKR fusion protein, more precisely to TNFR2-TWEAKR fusion protein acting as a double-antagonist to TNF-&#x3b1; and TWEAK, known as major causes of autoimmune arthritis which is one of autoimmune diseases. When the composition comprising TNFR2-TWEAKR fusion protein was treated to Th17 cells, the secretion of the inflammatory cytokine IL-17 was reduced but the secretion of the anti-inflammatory cytokine IL-10 generated in Treg cells was increased. Such effect of TNFR2-TWEAKR fusion protein was far greater than that of a single protein such as TNFR2-Fc or TWEAK-Fc. The TNFR2-TWEAKR fusion protein of the present invention has not only excellent treatment effect on arthritis in CIA mouse model not also excellent treatment effect on autoimmune rheumatoid arthritis by increasing the expression of Treg, the immune suppressive cells. Therefore, the TNFR2-TWEAKR fusion protein of the present invention can be effectively used as an active ingredient for the composition for the prevention and treatment of autoimmune disease.

TNF-A and TWEAK dual antagonist for the prophylaxis and treatment of autoimmune diseases

Growing community of inventors

Seoul, South Korea

Ho Youn Kim